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Prostaglandins (PGs) prevent damage of deeper structures due to an increase of mucus secretion, intensification of bicarbonate anions production (HCO3-), which neutralise acidic gastric content and the stimulation of mucosal phospholipids (6, 19, 20)

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If we simultaneously inhibited glucose and glutamine oxidation using a cocktail of etomoxir and UK5099, thus requiring cells to use fatty acids, the OCR change in uninfected cells was negligible (Fig

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